sstyphi 亂說話

概述
    河蜆又稱黃蜆、金蚶、扁螺等，廣泛分佈在我國湖泊、江河中，天然資源豐富。它們穴居於水底泥土表層，以浮游生物為食料，生長快，繁殖力強。除天然資源外，也適宜進行人工養殖。
    河蜆養殖既適合於湖泊大、中型水面放流增殖，也適合於小型水面或者池塘投飼、投肥養殖。養殖河蜆成本低、產量高，易捕撈，可以當年放養當年收穫，經濟效益顯著。 

第十八章　 斑馬魚與牛蛙實驗動物

一、斑馬魚

不說是草缸 因為稱不上是草缸說一尺 其實也沒有一尺 24*24*15沒有種太特別的東西 因為花不起太多的錢雖然開始養魚不過三個月光景 不過 還好我爬文爬得還算認真自己有兩個缸 一缸養孔雀球魚 一缸養燈魚水草幫別人設過兩個缸 在我又做了很多功課以後 發現一切都很快上手我承認 一開始真有許多小生命掛點在我無知的過去裡頭現在正在看文章的你 也許也正處在這個階段 在想著為什麼魚會一直死一開始 我跟很多人一樣 以為買了燈 缸 沙 過濾 魚 就可以了但是回來加了許多自來水 外掛放下去跑 高興地把魚給倒下去那天 一切都好好的 接著 魚死 水濁 才發現事情不是自己想的那樣開始作功課 很用功地爬文 哦~~ 原來有這麼多事情要注意呀!!!!
在我可以穩定地保持孔雀小缸的水質環境後 我設了第二個缸開始學著種水草 至少 到目前為止 都挺成功的 魚很快樂 蝦子一直生有了養魚的信心 恰巧有學弟想養魚 於是又重設缸開始把這樣的經驗跟大家分享 我想 其中一定有些地方不妥善 歡迎大家指正主要的出發點 是要相信設個缸並不像想像中的那麼花錢
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1.店名：李繼新疆牛肉拌麵
地址：台北市新生南路一段90號
電話：02-2393-1913

以下為提醒
CELLULAR & MOLECULAR BIOLOGY LETTERS
Volume 10, (2005) pp 101 – 121

Amyloid
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What makes a good PhD student?
Georgia Chenevix-Trench1
1. Georgia Chenevix-Trench is principal research fellow at the Queensland Institute of Medical Research, Royal Brisbane Hospital, Herston, Australia.

Cancer: Taming Puma

Les lysosomes
Les vésicules golgiennes ne servent pas toutes pour la sécrétion de protéines hors de la cellule. Certaines vésicules contiennent des enzymes digestives qui agissent à l'intérieur même de la cellule et qui ne peuvent être laissées libres dans le cytoplasme: on parle des lysosomes.
Du point de vue structural, les lysosomes sont de petites vésicules membraneuses de formes et de dimensions variables; une cellule peut en contenir plusieurs centaines. Les lysosomes contiennent un grand nombre d'enzymes différentes capables d'hydrolyser c'est-à-dire de digérer de grosses molécules comme des protéines, des glucides, des lipides, des acides nucléiques. Ces enzymes sont des hydrolases actives seulement à un pH de 5; ce pH acide du milieu lysosomial est maintenu grâce à des échanges actifs d'ions acides vers le milieu interne des lysosomes. La rupture accidentelle des membranes lysosomiales et le déversement des enzymes dans le cytoplasme est généralement sans conséquence puisque le pH du cytoplasme est neutre ce qui inactive alors les enzymes lysosomiales.

第一章：緒論
1、 填空題：
1、 細胞生物學是細胞整體、超微結構和分子水平上研究 及其 規律的科學。、

Abstract Annual Review of Physiology Vol. 60: 601-617 (Volume publication date March 1998) (doi:10.1146/annurev.physiol.60.1.601) CELL CYCLE REGULATION AND APOPTOSIS1 K. L. King* and ­J. A. Cidlowski#­ *Department of Molecular and Cellular Physiology, University of Cincinnati Medical Center, P. O. Box 670576, Cincinnati, Ohio 45267-0576; #National Institutes of Health, National Institute of Environmental Health Sciences, P.O. Box 12233 MD E2-02, Research Triangle Park, North Carolina 27709; e-mail: cidlowski@niehs.nih.gov Tissue homeostasis requires a balance between cell proliferation and death. Apoptosis and proliferation are linked by cell cycle regulators, and apoptotic stimuli affect both cell proliferation and death. Glucocorticoids induce G1 arrest and apoptosis in transformed lymphoid cells. Decreased expression of the cell cycle components c-myc and cyclin D3 is essential for glucocorticoid-induced growth arrest and death in dividing cells. Other G1 regulators, such as p53, pRb, and E2F, have also been implicated in apoptosis. Mice lacking either p53 or E2F display aberrant cell proliferation and tumor formation, suggesting that these proteins are involved in the elimination of abnormal cells through apoptosis. In contrast, pRb induces G1 arrest and suppresses apoptosis in cultured cells. Mice that lack pRb are nonviable and show ectopic mitosis and massive cell death, suggesting that pRb is an apoptotic suppressor. Further analysis of common components of apoptotic and cell cycle machinery may provide insight into the coordinated regulation of these antagonistic processes. 以上內容來自 Journal of Bioenergetics and Biomembranes (Historical Archive) Publisher: Springer Science+Business Media B.V., Formerly Kluwer Academic Publishers B.V. ISSN: 0145-479X (Paper) 1573-6881 (Online) DOI: 10.1007/BF00770028 Issue:  Volume 19, Number 5 Date:  October 1987 Pages: 427 - 442 Mini-Review Proton conductance through phospholipid bilayers: Water wires or weak acids? John Gutknecht1 (1)  Department of Physiology, Duke University, and Duke Marine Laboratory, 28516 Beaufort, North Carolina Received: 2 February 1987  Revised: 15 May 1987   Abstract  The proton/hydroxide (H+/OH–) permeability of phospholipid bilayer membranes at neutral pH is at least five orders of magnitude higher than the alkali or halide ion permeability, but the mechanism(s) of H+/OH– transport are unknown. This review describes the characteristics of H+/OH– permeability and conductance through several types of planar phospholipid bilayer membranes. At pH7, the H+/OH– conductances (G H/OH) range from 2–6 nS cm–2, corresponding to net H+/OH– permeabilities of (0.4–1.7)×10–5 cm sec–1. Inhibitors ofG H/OH include serum albumin, phloretin, glycerol, and low pH. Enhancers ofG H/OH include chlorodecane, fatty acids, gramicidin, and voltages >80 mV. Water permeability andG H/OH are not correlated. The characteristics ofG H/OH in fatty acid (weak acid) containing membranes are qualitatively similar to the controls in at least eight different respects. The characteristics ofG H/OH in gramicidin (water wire) containing membranes are qualitatively different from the controls in at least four different respects. Thus, the simplest explanation for the data is thatG H/OH in unmodified bilayers is due primarily to weakly acidic contaminants which act as proton carriers at physiological pH. However, at low pH or in the presence of inhibitors, a residualG H/OH remains which may be due to water wires, hydrated defects, or other mechanisms. Key Words  Proton conductance - proton permeability - phospholipid bilayer membrane - weak acid - water wire - fatty acid - gramicidin The references of this article are secured to subscribers. 以上內容來自